(A) Study of maximal oxygen uptake
Direct determination by traditional methods maximal oxygen uptake (VO2max), experimental process is extremely complex. 20th century 5O years, the famous Swedish exercise physiologist Ostrander (Astrand) pioneered indirect determination of maximal oxygen uptake nomogram method. Indirect determination of maximal oxygen uptake method is confirmed after making the application of this indicator has simple, economical, fast and so on. Studies have shown that maximal oxygen uptake is to evaluate the endurance athletes important indicator of bodily functions, asics womens running shoes the two have a strong positive correlation. The emergence of automatic gas analyzer, making the practice of using a direct method for the determination exercise maximal oxygen uptake become a reality. Also makes the maximal oxygen uptake of this indicator in sports science and practical application of more extensive and in-depth. Currently, the maximum oxygen uptake athlete research and application of exercise physiology is still an important issue.
(B) on the oxygen debt Rethinking Theory
Oxygen debt doctrine Hill (Hill), Meyer Hof (Meyerhof) and Magnus Leah (Margria) and others in the 1920s and 1930s created and perfect. asics gel kinsei 4 running shoes The traditional theory that oxygen debt: During intense exercise, because the body can not be provided to meet the needs of movement of oxygen, this time for the body to anaerobic metabolism, resulting in a large number of lactic acid, thus forming oxygen debt. In the recovery period still have to maintain a high body oxygen level to oxidation of lactic acid, oxygen debt repayment. Over the years oxygen debt theory was an important theoretical pillars of exercise physiology.
Since the mid-1980s, some exercise physiologists launched on oxygen debt, oxygen deficit and anaerobic threshold after these three concepts of controversy, causing more people to high-intensity exercise, asics gel noosa tri 7 running shoes whether in vivo hypoxia concern and interest . Pents (Bang), Brooks (Brooks), Gaisse (Gasser) and Khalida Adams (Haris) and others studies have shown that the human body engaged in high-intensity short-term recovery after exhaustive exercise early, blood lactate concentration continues to rise, and the oxygen consumption at this time already returned to resting levels; engaged in the process of prolonged exhaustive exercise blood lactate had already peaked, and in the subsequent course of the campaign gradually reduced, in continue to reduce the recovery period after exercise to the resting level, at which point the oxygen consumption was higher than the resting level. Demonstrated additional lactic acid and oxygen consumption after exercise no linear relationship. Thus proving the “oxygen debt” concept is not correct, put forward with “excess post-exercise oxygen consumption (EPOC)” concept.
(C) studies on individual lactate threshold
Human movement, asice gel noosa tri 8 runnning shoes as the exercise intensity increases, blood lactate levels will continue to rise, when the exercise intensity is increased to maximal oxygen uptake of about 60%, significantly higher blood lactate begins, the blood lactate inflection point is called as “anaerobic threshold.” Submaximal load exercise, the muscle tissue due to lack of oxygen causes anaerobic lactic acid is produced broad theory foundation. Which means that the muscles begin to hypoxia inflection point from aerobic to anaerobic energy to begin the transition. But there is much evidence that, at submaximal exercise, oxygen is not the real reason for the muscles produce lactic acid, so some exercise physiologist suggested to use lactate threshold instead of the anaerobic threshold concept.
Lactate threshold (LAT) concept is based on changes in blood lactate concentration and exercise intensity changes in the relationship concerned. clearance asics shoes When exercise intensity gradually increased, blood lactate changes occur two nonlinear inflection point, namely: when the blood lactate is 2mmol / L (mmol / l) occurs around the first inflection point; when 4mmol / L occurs around the second an inflection point. Currently, widely used at home and abroad blood lactate was 4mmol / L as the lactate threshold.
Stegrmann and Kinderman, who believe that lactic acid threshold without considering motion dynamics of the individual features. Because there is a big blood lactate inflection individual differences, they are based on exercise and post-exercise blood lactate kinetics characteristics, determined lactate threshold for each subject, saying this is the individual lactate threshold (ILAT), which proposed individual lactate threshold concept. asics gel kinsei 4 mens shoe
Because of individual lactate threshold improvement and relies on the improvement of maximal oxygen uptake, so it was decided to limit exercise capacity is an important indicator. Practice has proved that the individual lactate threshold intensity of the training is to improve exercise capacity limit of the best means to this end by the majority of coaches and athletes of all ages. Recently, studies of athletes in motion changes in individual lactate threshold, lactate threshold valve with self-guided exercise training has become the Exercise Physiology and Biochemistry of the important research topic.
(D) on exercise-induced fatigue research
Since 1880 Mosso (Mosso) began the study of human fatigue, exercise-induced fatigue on exercise physiology and sports medicine to become one of the core.
Kaposi Abramovich (Karpovich) the fatigue is defined as: “the work itself is the cause of fatigue is the phenomenon of decreased ability to work.” asics gel kinsei 4 womens shoe 1982 Fifth International Conference on Sport biochemistry, will fatigue is defined as: “the body’s physiological processes can not maintain its performance at a given level, and (or) can not maintain a predetermined intensity. “From the above definition can be seen, fatigue is a function of the body’s overall level of work efficiency or physiological phenomenon, should exercise training with the disease, and overtraining phase difference. Exercise-induced fatigue is a particularly complex physiological processes. It is caused by movement of multiple organ system functional changes and the combined result. asics gel kinsei 4 t139n Therefore, we must from the perspective of the whole body to consider this issue.
Exercise Fatigue Fatigue can be divided into central and peripheral fatigue. And then from the hub to the skeletal muscle cells within the cell metabolism process, in the middle or a part of any comprehensive changes in these processes, can cause fatigue. Recently proposed control chain or exercise-induced fatigue fatigue catastrophe theory, are trying to illustrate various aspects of sports fatigue causes, so exercise fatigue theory better for a more scientific approach to grasp eliminate fatigue.
Currently, Exercise Fatigue from mere knowledge of the mechanisms of energy consumption or accumulation of metabolites, the combined effects of multiple factors to the development of knowledge. asics t214n Research level also has cells, subcellular changes in the structure and function of biological molecules or ions down to the level.
(E) on the effects of exercise on free radical metabolism studies
Harman in 1956 on the basis of molecular biology radical theory proposes that the conduct in vivo metabolic process will produce some by-products, these byproducts called free radicals. Also known as free radical radical, means the outer track comprising one or more unpaired electrons of molecules, atoms, ions, or groups.
Dillard and Davies et al study confirmed that acute strenuous exercise can increase the concentration of free radicals. asics triathlon shoes Free radicals may be linked to the following factors: First, the intense exercise to strengthen the body’s metabolic processes, increased production of oxygen free radicals; followed by vigorous exercise, the accumulation of lactic acid inhibits free radical scavenging activity of the enzyme, the radical clearance rate; third movement of the body due to some substances can be automatically generated radical oxidation, such as catecholamines, reduced cytochrome C, hemoglobin, myoglobin, and other substances in strenuous exercise can be automatically generated by oxidation of free radicals .
Exercise-induced free radicals content increases, will lead to strengthening of lipid peroxidation, while the tissue and cell damage. asics gel kinsei 4 These injuries manifested in the following aspects: ① sports anemia and hemoglobinuria. Strenuous exercise erythrocyte oxyhemoglobin autoxidation rate strengthened in order to produce large amounts of free radicals, the red blood cell membrane fatty substances peroxidation, reducing the cell membrane deformability, fragility, leading to hemolysis, hemoglobinuria eventually occur and sports anemia. ② cause muscle fatigue. After strenuous exercise, excess free radicals can attack muscle fiber membrane and the sarcoplasmic reticulum, thus compromising its integrity, resulting in a number of ion transport and metabolism disorders. asics gel noosa tri 7 men shoes Alternatively, you can make the mitochondrial respiratory chain is damaged, so that barriers to the formation of ATP, resulting in decreased ability of muscle work to accelerate the development of the fatigue process. ③ delayed onset muscle soreness. At present, evidence suggests that delayed onset muscle soreness and free radical damage.
Studies have shown that aerobic exercise can increase the body’s antioxidant enzyme activity during exercise can effectively remove excess free radicals produced. Also you can add expatriate antioxidants, such as vitamin E, vitamin C, and some medicine can effectively improve the body antioxidant capacity.
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